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NAT8L Knockout T47D Polyclonal Cells
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NAT8L Knockout T47D Polyclonal Cells

Cat. No. ARG11957
Product Type:

Polyclonal Cell Population

Species:

Homo sapiens (Human)

Tissue Source:

Breast (mammary gland)

Growth Properties:

Adherent

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Short Description 🔒

NAT8L Knockout T47D Polyclonal Cells is a knockout polyclonal cell population. The NAT8L was knocked out in the T47D cells using the CRISPR RNP technique.

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Product Details
Cell Engineering
Immortalization
Culture Conditions
Quality Control
Disclaimer

Product Details

Product Type:
Polyclonal Cell Population
Species:
Homo sapiens (Human)
Tissue Source:
Breast (mammary gland)
Disease:
Ductal carcinoma
Morphology:
Epithelial-like
Growth Mode:
Adherent
Age:
54 years
Sex of Donor:
Female
Derived From Site:
Metastatic; Pleural effusion
Size/Quantity:
1 million
Shipping info:
Cryopreserved in vials and shipped on dry ice
Storage:
Liquid nitrogen (LN2)

Cell Engineering Information

Host Cell:
T-47D
Gene Name:
NAT8L
Gene Identifier:
NCBI Gene ID 339983

Immortalization Information

No immortalization information available.

Culture Conditions

Growth medium:
RPMI 1640
Supplement(s):
10% Fetal Bovine Serum, 10μg/mL Insulin, 1% Penicillin-Streptomycin Solution
Temperature:
37°C
Atmosphere:
5% CO₂

Quality Control

Mycoplasma testing:
Negative for mycoplasma through PCR analysis
Sterility testing:
The bacterial, yeast, and fungi are not detected in these cells by daily monitor.

Disclaimer

Intended Use:
This product is intended for laboratory in vitro use only. It is not intended for diagnostic, therapeutic, or clinical applications.
Disclaimer:
Ascent Research endeavors to provide accurate and up-to-date product information. However, no warranties or representations are made regarding its completeness or reliability.
Usage:
By accepting this product, the customer acknowledges and agrees to assume all risks associated with its receipt, handling, storage, disposal, and use. This product is provided "AS IS".

Description 🔒

Complete description is available upon request.

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